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In the resolution phase of acute lung injury, extensive apoptosis of type II pneumocytes is largely responsible for the disappearance of these cells. The time frame within which the apoptotic response occurs is variable and is likely to be dependent upon the specific etiology and extent of the injury. Apoptosis is a form of programmed cell death that results in the orderly and efficient removal of damaged cells, such as those resulting from DNA damage or during development. Apoptosis can be triggered by signals from within the cell, such as genotoxic stress, or by extrinsic signals, such as the binding of ligands to cell surface death receptors. Apoptosis is a way for the body to keep checks and balances on the natural cell division process of mitosis or continued cell growth and regeneration. Why Cells Undergo Apoptosis There are several instances in which cells may need to self-destruct. In some situations, cells may need to be removed to ensure proper development.

Apoptosis (from Ancient Greek ἀπόπτωσις, apóptōsis, "falling off") is a form of programmed cell death that occurs in multicellular organisms. Biochemical events lead to characteristic cell changes ( morphology) and death. These changes include blebbing, cell shrinkage, nuclear fragmentation, chromatin condensation, chromosomal DNA fragmentation, and global mRNA decay. We conclude that proliferation of type II pneumocytes occurs during the early phase of acute lung injury and is of variable extent and duration. In the resolution phase of acute lung injury, extensive apoptosis of type II pneumocytes is largely responsible for the disappearance of these cells. The time frame within which the apoptotic response occurs is variable and is likely to be dependent upon the specific etiology and extent of the injury. Apoptosis is a form of programmed cell death that results in the orderly and efficient removal of damaged cells, such as those resulting from DNA damage or during development.

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bab.la is not responsible for their content. EnglishWe "induce apoptosis" in Swedish.

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In vertebrates, apoptosis is important for proper development, maintenance of tissue homeostasis.

Yen-shen Lu. Chi-Hsin Lin. Chi-Mei Hsueh. Yen-shen Lu. We further confirmed that apoptosis score was linearly associated with thrombotic score. These results suggest that endothelial apoptosis may be an independent risk factor for thrombosis.

We conclude that proliferation of type II pneumocytes occurs during the early phase of acute lung injury and is of variable extent and duration.
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This is the currently selected item. Sort by: Top Voted. Apoptosis. Biology is brought to you with support from the Amgen Foundation. Published: 01 April 2004 LDFF, the large molecular weight DNA fragmentation factor, is responsible for the large molecular weight DNA degradation during apoptosis in Xenopus egg extracts. Zhi Gang Our results showed that a significantly high level of reactive oxygen species was induced but not responsible for apoptosis in breast cancer cells by FAS inhibition. Instead, NADPH accumulation resulting from FAS inhibition was found to stimulate NADPH oxidase to generate reactive oxygen species and highly associated with apoptosis induction.

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dysplasia, and conduction disturbances that may be responsible for sudden death. Sep 24, 2013 We identified Eiger, the Drosophila tumor necrosis factor (TNF) homolog, as the signal responsible for apoptosis-induced apoptosis (AiA). Feb 23, 2011 Programmed cell death called apoptosis serves a healthy and Scientists believe that too much apoptosis is at least partly to blame for some Apoptosis is a process of programmed cell death that occurs in multicellular organisms. Fas associated via death domain (FADD) protein is responsible for the pases are responsible for the cleavage of different substrates during apoptosis.

”Omics” of lipi Visa mer. 3,90 MSEK Budget. effort in the field of programmed cell death (also known as apoptosis).